Drug Discovery Opinion

Researchers at the University of Warwick have suggested that eating broccoli could undo the damage caused to heart blood vessels by diabetes. They found that sulforaphane, a compound found in broccoli, can prevent biochemical dysfunction induced by hyperglycemia in cultured endothelial cells. People with diabetes have a particularly high risk of heart disease and stroke, which are linked to damaged blood vessels.

Sulforaphane may also trigger production of thioredoxin, which protects against cell damage in the heart.

Studies have also linked consumption of cruciferous vegetables, such as broccoli, with decreased incidence of ischemic stroke.

It has also been reported that 3,3′-diindolylmethane and indole-3-carbinol, other compounds found in cruciferous vegetables, may have anticancer, antiviral, antibacterial and antioxidant properties.

Bookmark It

Hide Sites

Related posts:

1. Man or Mouse – It’s Important in Diabetes Research During the development of type 2 diabetes, uptake of glucose from the blood by muscle and fat cells in response...
2. You Are What You Eat The link between diet and epigenetics in humans is notoriously difficult to study since people are, unsurprisingly, resistant to being...
3. Non-peptide GLP-1 Agonist Shows Efficacy in Mouse Model of Diabetes Metabolic syndrome is a combination of medical disorders that increase the risk of developing cardiovascular disease, diabetes and obesity. A...
4. New DPP-4 Inhibitor Demonstrates Glucose Control in Type 2 Diabetes Type-2 diabetes is a metabolic disorder that is increasing rapidly in the developed world. The disease is caused by reduced...
5. New Insights into Adipogenesis The nuclear receptor, PPARγ, has hitherto been regarded as the master regulator of adipogenesis, without which new adipose tissue cannot...

Related posts brought to you by Yet Another Related Posts Plugin.

 Email This Post

This entry was posted on Monday, August 11th, 2008 at 8:41 am and is filed under News. You can follow any responses to this entry through the RSS 2.0 feed. You can leave a response, or trackback from your own site.